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Plant Disease · Plant disease

Septoria Tritici Blotch

Zymoseptoria tritici (teleomorph Mycosphaerella graminicola)

Also known as: Septoria leaf blotch, STB, Speckled leaf blotch

Septoria tritici blotch is a foliar fungal disease of wheat caused by Zymoseptoria tritici. It produces irregular tan lesions studded with small dark fruiting bodies and is one of the most damaging leaf diseases of wheat in temperate, humid regions.

Dated referenceLast reviewed: 2026-07-13Updated: 2026-07-13
Illustrative diagram · AgricultureID (original)

Septoria tritici blotch is caused by the fungus Zymoseptoria tritici and is among the most economically important leaf diseases of wheat in cool, wet climates such as north-western Europe. It attacks the leaves, gradually reducing the healthy green area that the plant needs to fill grain, and is most damaging when it reaches the upper leaves during stem elongation and heading.

A distinctive feature of the disease is the presence of tiny dark fruiting bodies, called pycnidia, embedded within the dead leaf tissue. The pathogen has a relatively long latent period between infection and visible symptoms, which makes early detection difficult and means disease can be building up in a crop before it becomes obvious.

Symptoms and identification

Symptoms usually begin as small chlorotic (yellowing) flecks that develop into irregular, tan-to-brown blotches with a somewhat angular outline, often bounded by leaf veins. As the lesions age, numerous small dark pycnidia become visible within the dead tissue, giving it a speckled or peppered appearance that is the most reliable field diagnostic feature.

Pathogen and disease cycle

Zymoseptoria tritici survives between crops on infected wheat residue and on volunteer plants. From this residue it produces wind-dispersed sexual spores (ascospores) that can initiate infections over distance, while asexual spores (pycnidiospores) formed in pycnidia are moved shorter distances by rain splash within and between plants, driving the disease up the canopy.

  • Infected crop residue and volunteer wheat carry the fungus between seasons
  • Wind-dispersed ascospores can start new infections across a region
  • Rain-splashed pycnidiospores move the disease from lower to upper leaves
  • A long latent period allows infection to build before symptoms appear

Hosts and favourable conditions

Wheat is the principal host, with triticale also affected to a lesser degree. Disease pressure is greatest in cool, humid regions where rainfall keeps the canopy wet and provides the splash events the pathogen needs to spread upward. Early sowing, dense canopies, and susceptible varieties all tend to increase risk.

  • Cool, wet, humid weather with frequent rainfall and prolonged leaf wetness
  • Early autumn sowing that exposes crops to more infection cycles
  • Dense, lush canopies that stay wet and favour splash dispersal
  • Susceptible varieties and short rotations that keep inoculum high

Management principles

Management is built on integrated tactics that reduce inoculum and slow the disease reaching the upper leaves, rather than on any single measure. Resistant varieties are the foundation, supported by cultural practices and, where justified, protection of the upper canopy under locally authorized guidance.

  • Choose varieties with good resistance ratings for the local region
  • Use crop rotation and residue management to reduce carry-over inoculum
  • Consider later sowing where agronomically suitable to reduce early infection
  • Avoid excessive nitrogen that produces dense, humid canopies
  • Monitor upper leaves closely from stem elongation through heading

Relationships

Evidence-backed connections in the knowledge graph.

Scope & limitations

Geographic scope: Global; most damaging in cool, humid wheat-growing regions such as north-western Europe, with severity varying by season, variety, and local rainfall.

Climate context: Favoured by cool, wet, humid conditions with frequent rain splash that disperses spores up the canopy; a long latent period lets infections build before symptoms show.

  • This is a general overview and does not replace field diagnosis or region-specific extension guidance.
  • Pathogen populations and their sensitivity to control-product classes vary by region and change over time; consult local authorities for current status.
  • Control-product recommendations are jurisdiction-specific and are deliberately not provided here.

Sources

This article draws on the following authoritative sources. See our sources & methodology for how they are selected.

  1. [1]CABI Digital Library — Crop Protection Compendium (opens in a new tab)

    CABI (Centre for Agriculture and Bioscience International)

    High

    Cited for: Pathogen biology, disease cycle, and host range

    Type:
    Reference database
    Jurisdiction:
    Global
    Accessed:
    2026-07-12
  2. [2]EPPO Global Database (opens in a new tab)

    European and Mediterranean Plant Protection Organization (EPPO)

    Authoritative

    Cited for: Pathogen nomenclature and distribution

    Type:
    Reference database
    Jurisdiction:
    Global
    Accessed:
    2026-07-12
  3. [3]AHDB — Agriculture and Horticulture Development Board (opens in a new tab)

    Agriculture and Horticulture Development Board (AHDB)

    High

    Cited for: Wheat disease identification and integrated management principles

    Type:
    Government agency
    Jurisdiction:
    United Kingdom
    Accessed:
    2026-07-12