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Plant Disease · Plant disease

Ergot

Claviceps purpurea (and related Claviceps species)

Also known as: Ergot of rye, Ergot of cereals

Ergot is a fungal disease of open-flowering cereals and grasses, caused by Claviceps purpurea, in which dark sclerotia replace developing grain in infected florets. It is favoured by cool, wet weather during flowering and is a recognised grain-quality and food-safety concern because of the alkaloids sclerotia contain.

Dated referenceLast reviewed: 2026-07-12Updated: 2026-07-12
Ergot on a cereal head
The British Museum, Ian Richardson, 2015-09-03 09:32:52, via Wikimedia Commons (CC BY 2.0) CC BY 2.0 (opens in a new tab) source (opens in a new tab)Downloaded from Wikimedia Commons; resized to a maximum width of 1280 px and recompressed as JPEG for web delivery. Content unaltered.Illustrative only — visual symptoms or appearance alone may not be sufficient for reliable identification.

Ergot is caused by Claviceps purpurea, a fungus that infects the flowers of rye, wheat, triticale, and many other grasses, replacing the developing grain with a hard, dark resting structure called a sclerotium. Rye is particularly susceptible because of its extended open-flowering habit and cross-pollination.

Beyond the direct yield effect of lost grain, ergot sclerotia contain alkaloids that are toxic to humans and livestock if consumed in sufficient quantity, making grain contamination a food- and feed-safety concern in addition to an agronomic one.

Identification

Infected florets first exude a sticky, sweet-smelling fluid known as honeydew, which contains fungal spores and can attract insects. This stage is followed by the development of a hard, dark purplish-black sclerotium in place of the normal kernel, often protruding noticeably beyond the surrounding grain.

Biology and disease cycle

Sclerotia that fall to the soil, or that are returned with contaminated seed, can survive and germinate the following season to produce small, mushroom-like structures that release airborne ascospores. These spores infect open florets during flowering, and secondary spread can occur through insects and rain splash carrying the honeydew stage between florets.

  • Sclerotia can persist in soil or as a grain contaminant between seasons
  • Infection requires open flowers, so crops and grasses with an extended flowering period are more exposed
  • Nearby infected grasses can serve as an additional source of spores

Affected hosts

Rye is the most susceptible major cereal because of its extended open-flowering and cross-pollinating habit. Wheat and triticale can also be affected, with triticale generally more exposed than wheat because it shares some of rye's flowering characteristics. Many wild and forage grasses can also host the fungus.

Risk factors

  • Cool, wet weather during the flowering period
  • An extended or uneven flowering period, including in some hybrid seed production systems
  • Nearby infected grasses or previous ergot-contaminated seed
  • Reduced or delayed pollination that leaves florets open longer

Prevention and monitoring

Because ergot spreads through open flowers and persists as sclerotia, prevention combines clean planting material, field sanitation, and post-harvest grain cleaning.

  • Use certified, ergot-free seed
  • Rotate away from highly susceptible cereals and control grassy weeds and volunteer plants that can host the fungus
  • Bury sclerotia through tillage where consistent with the local cropping system
  • Clean harvested grain to remove sclerotia before use as food or feed

Relationships

Evidence-backed connections in the knowledge graph.

Scope & limitations

Geographic scope: Global; ergot occurs wherever susceptible cereals and grasses are grown, with rye-growing regions historically most affected.

Climate context: Favoured by cool, wet weather during flowering, which prolongs floret exposure and favours spore germination and infection.

  • This is a general overview and does not replace field diagnosis or region-specific extension guidance.
  • Food- and feed-safety thresholds for ergot contamination are set by local or national authorities and are not provided here.
  • Control-product recommendations are jurisdiction-specific and are deliberately not provided here.

Sources

This article draws on the following authoritative sources. See our sources & methodology for how they are selected.

  1. [1]CABI Digital Library — Crop Protection Compendium (opens in a new tab)

    CABI (Centre for Agriculture and Bioscience International)

    High

    Cited for: Pathogen biology, disease cycle, and host range

    Type:
    Reference database
    Jurisdiction:
    Global
    Accessed:
    2026-07-12
  2. [2]EPPO Global Database (opens in a new tab)

    European and Mediterranean Plant Protection Organization (EPPO)

    Authoritative

    Cited for: Pathogen nomenclature and distribution

    Type:
    Reference database
    Jurisdiction:
    Global
    Accessed:
    2026-07-12
  3. [3]Cornell CALS — Plant pathology and crop resources (opens in a new tab)

    Cornell University College of Agriculture and Life Sciences

    High

    Cited for: Cereal disease risk factors and sanitation guidance

    Type:
    University extension service
    Jurisdiction:
    United States (New York)
    Accessed:
    2026-07-12
  4. [4]Iowa State University Extension and Outreach (opens in a new tab)

    Iowa State University Extension and Outreach

    High

    Cited for: Field-level identification and grain-quality guidance

    Type:
    University extension service
    Jurisdiction:
    United States (Iowa)
    Accessed:
    2026-07-12